Search results for "Parvovirus H-1"

showing 7 items of 7 documents

Oncolytic Virotherapy as Emerging Immunotherapeutic Modality: Potential of Parvovirus H-1

2014

Human tumors develop multiple strategies to evade recognition and efficient suppression by the immune system. Therefore, a variety of immunotherapeutic strategies have been developed to reactivate and reorganize the human immune system. The recent development of new antibodies against immune check points may help to overcome the immune silencing induced by human tumors. Some of these antibodies have already been approved for treatment of various solid tumor entities. Interestingly, targeting antibodies may be combined with standard chemotherapy or radiation protocols. Furthermore, recent evidence indicates that intratumoral (it) or intravenous (iv) injections of replicative oncolytic viruse…

Cancer ResearchParvovirus H-1medicine.medical_treatmentautonomous parvovirusReview Articlelcsh:RC254-282JX-594Immune systemAntigenmedicineDentritic cellsdendritic cellsVirotherapybusiness.industryImmunotherapylcsh:Neoplasms. Tumors. Oncology. Including cancer and carcinogensOncolytic virusH-1PVT-VECtalimogene laherparepvecOncologyCTLA-4ImmunologyCTLA-4immunotherapyTalimogene laherparepvecbusinessFrontiers in Oncology
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Effective infection, apoptotic cell killing and gene transfer of human hepatoma cells but not primary hepatocytes by parvovirus H1 and derived vector…

2001

Autonomous parvoviruses preferentially replicate in and kill in vitro–transformed cells and reduce the incidence of spontaneous and implanted tumors in animals. Because of these natural oncotropic and oncolytic properties, parvoviruses deserve to be considered as potential antitumor vectors. Here, we assessed whether parvovirus H1 is able to kill human hepatoma cells by induction of apoptosis but spares primary human liver cells, and whether the former cells can efficiently be transduced by H1 virus–based vectors. Cell death, infectivity, and transgene transduction were investigated in Hep3B, HepG2, and Huh7 cells and in primary human hepatocytes with natural and recombinant H1 virus. All h…

Cancer ResearchProgrammed cell deathCarcinoma HepatocellularParvovirus H-1Cell SurvivalvirusesGenetic VectorsApoptosisVirus ReplicationVirusCell LineParvoviridae InfectionsParvovirusTransduction (genetics)Transduction GeneticTumor Cells CulturedHumansMolecular BiologybiologyParvovirusLiver NeoplasmsGene Transfer Techniquesbiology.organism_classificationVirologyMolecular biologydigestive system diseasesOncolytic virusCell killingApoptosisDNA ViralHepatocytesMolecular MedicineCancer gene therapy
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Activation of the human immune system via toll-like receptors by the oncolytic parvovirus H-1.

2012

This study aimed to investigate the function of toll-like receptors (TLRs) during oncolytic parvovirus H-1 (H-1PV)-induced human immune responses. First, the role of TLRs in the activation of the NFκB transcription factor was characterized; second, the immunologic effects of H-1PV-induced tumor cell lysates (TCL) on human antitumor immune responses were evaluated. A human ex vivo model was used to study immune responses with dendritic cells (DCs). Human embryonic kidney cells (HEK293) transfected to stably express TLRs were used as potential human DC equivalents to further investigate the role of specific TLRs during immune activation. TLR3 and TLR9 were activated by H-1PV infection, which …

Cytotoxicity ImmunologicH-1 parvovirusCancer ResearchCytoplasmParvovirus H-1chemical and pharmacologic phenomenaEnzyme-Linked Immunosorbent AssayBiologyKidneyProinflammatory cytokineParvoviridae InfectionsImmune systemTumor Cells CulturedHumansMelanomaCells CulturedCell NucleusOncolytic VirotherapyTumor Necrosis Factor-alphaToll-Like ReceptorsNF-kappa BDendritic CellsAcquired immune systemFlow CytometryCell biologyOncolytic virusOncolytic VirusesOncologyImmune SystemImmunologyTLR3CytokinesTumor necrosis factor alphaSignal transductionSignal TransductionInternational journal of cancer
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Regression of advanced rat and human gliomas by local or systemic treatment with oncolytic parvovirus H-1 in rat models

2010

Oncolytic virotherapy is a potential treatment modality under investigation for various malignancies including malignant brain tumors. Unlike some other natural or modified viruses that show oncolytic activity against cerebral neoplasms, the rodent parvovirus H-1 (H-1PV) is completely apathogenic in humans. H-1PV efficiently kills a number of tumor cells without harm to corresponding normal ones. In this study, the concept of H-1PV-based virotherapy of glioma was tested for rat (RG-2 cell-derived) and for human (U87 cell-derived) gliomas in immunocompetent and immunodeficient rat models, respectively. Large orthotopic rat and human glioma cell-derived tumors were treated with either single …

H-1 parvovirusCancer ResearchPathologymedicine.medical_specialtyParvovirus H-1Secondary infectionAntibodies ViralPolymerase Chain ReactionVirusGliomamedicineAnimalsHumansVirotherapyOncolytic VirotherapybiologyBrain NeoplasmsParvovirusBrainGliomamedicine.diseasebiology.organism_classificationAntibodies NeutralizingMagnetic Resonance ImagingXenograft Model Antitumor AssaysRatsOncolytic virusDisease Models AnimalOncologyViral replicationBasic and Translational InvestigationsDNA ViralNeurology (clinical)Neuro-Oncology
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Killing of p53-deficient hepatoma cells by parvovirus H-1 and chemotherapeutics requires promyelocytic leukemia protein

2008

To evaluate the synergistic targeting and killing of human hepatocellular carcinoma (HCC) cells lacking p53 by the oncolytic autonomous parvovirus (PV) H-1 and chemotherapeutic agents and its dependence on functional promyelocytic leukemia protein (PML).The role of p53 and PML in regulating cytotoxicity and gene transfer mediated by wild-type (wt) PV H-1 were explored in two pairs of isogenic human hepatoma cell lines with different p53 status. Furthermore, H-1 PV infection was combined with cytostatic drug treatment.While the HCC cells with different p53 status studied were all susceptible to H-1 PV-induced apoptosis, the cytotoxicity of H-1 PV was more pronounced in p53-negative than in p…

H-1 parvovirusLiver CancerH-1 parvovirusCarcinoma HepatocellularParvovirus H-1virusesAntineoplastic AgentsApoptosisPromyelocytic Leukemia ProteinPromyelocytic leukemia proteinDrug TherapyCell Line TumorHumansNuclear proteinCytotoxicityMembrane Potential MitochondrialbiologyParvovirusTumor Suppressor ProteinsLiver NeoplasmsGastroenterologyvirus diseasesNuclear ProteinsGeneral Medicinebiology.organism_classificationCombined Modality Therapydigestive system diseasesOncolytic virusApoptosisCancer researchbiology.proteinFluorouracilCisplatinTumor Suppressor Protein p53Transcription FactorsWorld Journal of Gastroenterology
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Influence of the oncolytic parvovirus H-1, CTLA-4 antibody tremelimumab and cytostatic drugs on the human immune system in a human in vitro model of …

2013

Bernd Heinrich,* Katrin Goepfert,* Maike Delic, Peter R Galle, Markus MoehlerUniversity Medical Center of the Johannes Gutenberg University Mainz, 1st Department of Internal Medicine, Langenbeckstrasse, Mainz, Germany *These authors contributed equally to this workIntroduction: Tumor-directed and immune-system-stimulating therapies are of special interest in cancer treatment. Here, we demonstrate the potential of parvovirus H-1 (H-1PV) to efficiently kill colorectal cancer cells and induce immunogenicity of colorectal tumors by inducing maturation of dendritic cells (DCs) alone and also in combination with cytostatic drugs in vitro. Using our cell culture model, we have additionally investi…

Parvovirus H-1business.industrymedicine.medical_treatmentOncoTargets and TherapyOncolytic virusImmune systemCytokineOncologyAntigenCTLA-4ImmunologyCancer researchmedicineSW480Cytotoxic T cellPharmacology (medical)dendritic cellsbusinessTremelimumabmedicine.drugOriginal ResearchOncoTargets and Therapy
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Parvovirus H-1-Induced Tumor Cell Death Enhances Human Immune Response In Vitro via Increased Phagocytosis, Maturation, and Cross-Presentation by Den…

2005

Oncotropic and oncolytic viruses have attracted high attention as antitumor agents because they preferentially kill cancer cells in vitro and reduce the incidence of spontaneous, induced, or implanted animal tumors. Some autonomous parvoviruses (H-1, minute virus of mice) and derived recombinant vectors are currently under preclinical evaluation. Still not fully understood, their antitumor properties involve more than just tumor cell killing. Because wild-type parvovirus-mediated tumor cell lysates (TCLs) may trigger antigen-presenting cells (APCs) to augment the host immune repertoire, we analyzed phagocytosis, maturation, and crosspresentation of H-1-induced TCLs by human dendritic cells …

Skin NeoplasmsParvovirus H-1ApoptosisBiologyParvovirusMiceImmune systemCross-PrimingAntigenPhagocytosisAntigens NeoplasmHLA-A2 AntigenTumor Cells CulturedGeneticsCytotoxic T cellAnimalsHumansMelanomaMolecular BiologyCryopreservationCross-presentationCell DifferentiationDendritic cellDendritic CellsOncolytic virusCancer cellImmunologyCancer researchMolecular MedicineT-Lymphocytes CytotoxicHuman Gene Therapy
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